Dual pathways of carbon monoxide-mediated vasoregulation: modulation by redox mechanisms.
Circulation Research (2009)
- PubMed: 19745167
Available from www.ncbi.nlm.nih.gov
or
Abstract
Vascular tissues produce carbon monoxide (CO) via HO-dependent and HO-independent mechanisms; the former in tandem with biliverdin and iron and the latter as a lone product. CO has been shown to function as both a vasoconstrictor and vasodilator; however, factors that dictate the vasoregulatory phenotype of this gas are unknown.
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