Peripheral and central activation of trigeminal pain pathways in migraine: Data from experimental animal models

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Abstract

EEG-studies in migraine in the last decade has contributed modestly to the understanding of headache pathogenesis. Headache patient groups seem to have increased EEG responses to photic stimulation, but a useful biological marker for migraine in single patients has not been found. In future EEG and QEEG studies we recommend to use follow-up designs and record several EEGs across the migraine cycle. It is also important to use a blinded study design in order to avoid selection bias. A clinical EEG should be performed in patients with acute headache attacks when either epilepsy, basilar migraine, migraine with prolonged aura or alternating hemiplegia is suspected. Unequivocal epileptiform abnormalities usually suggest a diagnosis of epilepsy. In children with occipital spike-wave activity the probable diagnosis is childhood epilepsy with occipital paroxysms (CEOP). The final diagnosis of either an epilepsy syndrome or migraine must be mainly based on a clinical judgement.

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Buzzi, M. G., Tassorelli, C., & Nappi, G. (2003). Peripheral and central activation of trigeminal pain pathways in migraine: Data from experimental animal models. Cephalalgia, Supplement. Blackwell Publishing Ltd. https://doi.org/10.1046/j.1468-2982.23.s1.1.x

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