In more recent times, the global spread of HIV-1, outbreaks related to avian influenza, and the emergence of severe acute respiratory syndrome and Middle Eastern respiratory syndrome, to name a few, profoundly illustrate how an infection can spread worldwide in a very rapid fashion (Table I).1 In this issue of the Journal, Kampmann et al2 and Lindblad et al3 describe the ongoing Ebola virus disease (EVD) outbreak that is currently ravaging West Africa, and Petidemange et al4 describe how the immune system might be promoting the viral spread and immunopathology of chikungunya virus (CHIKV) infection. [...]patients with atopic dermatitis vaccinated transcutaneously have reduced neutralization antibodies to yellow fever virus that correlate with their serum IgE levels.14 More recently, intradermal Fluzone (Sanofi Pasteur, Swiftwater, Pa) was found to induce lower influenza hemagglutination inhibition antibody titers in patients with atopic dermatitis colonized with Staphylococcus aureus.15 Lastly, research and development of vaccines and novel therapies for these types of infections are important and might be the only long-term solution to prevent further spread.
Barron, M. A., & Leung, D. Y. M. (2015, April 1). Lessons from Ebola and readiness for new emerging infectious threats. Journal of Allergy and Clinical Immunology. Mosby Inc. https://doi.org/10.1016/j.jaci.2015.01.007