Gene expression profiling studies reported by Hurt et al. in this issue of Cancer Cell reveal an unexpectedly frequent overexpression of c-maf in multiple myeloma and identify molecular targets of c-maf transactivation. The results define c-maf as a prototype of a class of oncogenes that not only stimulate cell cycle progression, but also promote pathological interactions between tumor and stroma cells.
CITATION STYLE
Kienast, J., & Berdel, W. E. (2004). c-maf in multiple myeloma: An oncogene enhancing tumor-stroma interactions. Cancer Cell. Cell Press. https://doi.org/10.1016/S1535-6108(04)00030-3
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