This report describes the presentation and clinical course of a 40-year-old woman who had an emergency admission for eclampsia During routine investigations, she was found to have profound hypercalcaemia, the cause of which was identified as milk-alkali syndrome, caused by self-medication with antacid tablets for dyspepsia. Treatment with aggressive rehydration, bisphosphonates and discontinuation of antacid tablets restored normocalcaemia. The patient made a full recovery with no long-term side-effects. Her male infant was safely delivered with no deleterious effects of exposure to high calcium concentrations in utero.
CITATION STYLE
Bailey, C. S., Weiner, J. J., Gibby, O. M., & Penney, M. D. (2008). Excessive calcium ingestion leading to milk-alkali syndrome. Annals of Clinical Biochemistry, 45(5), 527–529. https://doi.org/10.1258/acb.2008.008006
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