Autophagy is required for mitochondrial function, lipid metabolism, growth, and fate of KRASG12D-driven lung tumors

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Abstract

FAO, fatty acid oxidation; FAs, fatty acids; GEMMs, genetically engineered mouse models; NSCLC, nonsmall- cell lung cancer; TDCLs, tumorderived cell lines; TCA, tricarboxylic acid. © 2013 Landes Bioscience.

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Guo, J. Y., & White, E. (2013). Autophagy is required for mitochondrial function, lipid metabolism, growth, and fate of KRASG12D-driven lung tumors. Autophagy, 9(10), 1636–1638. https://doi.org/10.4161/auto.26123

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