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Malignant malaria and microangiopathies: Merging mechanisms

Blood
DOI: 10.1182/blood-2009-12-254060
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Abstract

In this issue of Blood, Bridges and colleagues report that Plasmodium falciparum-infected erythrocytes are able to attach to the endothelial surface by binding platelet-decorated VWF strands in a CD36-dependent fashion, revealing a new mechanism for erythrocyte cytoadherence in malaria.

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López, J. A. (2010, February 18). Malignant malaria and microangiopathies: Merging mechanisms. Blood. American Society of Hematology. https://doi.org/10.1182/blood-2009-12-254060

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