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BBOX1 promotes triple-negative breast cancer progression by controlling IP3R3 stability

Molecular and Cellular Oncology (2020) 7(6)
DOI: 10.1080/23723556.2020.1813526
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Abstract

Limited therapeutic options impede the clinical outcome of triple-negative breast cancer (TNBC). Our recent study uncovered a novel signaling pathway implicating gamma-butyrobetaine hydroxylase 1 (BBOX1) in the control of cell growth in TNBC, via inositol 1, 4, 5-trisphosphate receptor type 3 (IP3R3) mediated calcium signaling which is essential for cellular energy metabolism.

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Liao, C., & Zhang, Q. (2020). BBOX1 promotes triple-negative breast cancer progression by controlling IP3R3 stability. Molecular and Cellular Oncology, 7(6). https://doi.org/10.1080/23723556.2020.1813526

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