Abstract
In the past 20 years, it has become clear that reactive oxygen species (ROS) contribute to the development of hypertension via myriad effects. While ROS are essential for normal cell function, they mediate pathological changes in the brain, the kidney and blood vessels associated with the genesis of chronic hypertension.
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Wu, J., & Harrison, D. G. (2015). Oxidative stress and hypertension. In Blood Pressure and Arterial Wall Mechanics in Cardiovascular Diseases (Vol. 9781447151982, pp. 175–191). Springer-Verlag London Ltd. https://doi.org/10.1007/978-1-4471-5198-2_15
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