Abstract
Given a stable cardiac output (CO) and inspiratory oxygen concentration (FIO 2), any gas exchange abnormality leading to hypoxia or hypercapnia may be explained solely on the basis of an altered distribution of the ventilation and perfusion regardless of the underlying disease [1]. © 2006 Springer-Verlag Berlin Heidelberg.
Cite
CITATION STYLE
APA
Calzia, E., & Radermacher, P. (2006). Alveolar ventilation and pulmonary blood flow: The concept. In Applied Physiology in Intensive Care Medicine (pp. 21–24). Springer Berlin Heidelberg. https://doi.org/10.1007/3-540-37363-2_6
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