Abstract
In this issue of Blood, Elks et al describe a novel role for activated hypoxia inducible factor (HIF)-1α in sustaining inflammation by delaying neutrophilic retrograde emigration and preventing neutrophil apoptosis through the inhibition of prolyl hydroxylase (PHD) activity. This property is a novel function for HIFs, the master regulators of our body's response to hypoxia.
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CITATION STYLE
APA
Eubank, T. D., & Marsh, C. B. (2011, July 21). HIFs: A-cute answer to inflammation? Blood. American Society of Hematology. https://doi.org/10.1182/blood-2011-05-355164
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