Transgenic knockin mice expressing a common loss-of-function mutation in human TET2 exhibit aging-related accelerated myeloid leukemia development and skewing of myelopoiesis toward the production of proinflammatory MHC-IIhi monocytes that may contribute to disease.
CITATION STYLE
Pietras, E. M., & Degregori, J. (2022). Dangerous Liaisons between Tet2 Mutation, Inflammatory Monocytes, and Leukemogenesis. Cancer Discovery, 12(10), 2234–2236. https://doi.org/10.1158/2159-8290.CD-22-0846
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