Abstract
In comparison to wild-type herpesvirus saimiri, viral interleukin-17 gene knockout mutants have unaltered behavior regarding viral replication, T-cell transformation in vitro, and pathogenicity in cottontop tamarins. Thus, this gene is not required for T-cell lymphoma induction but may contribute to apathogenic viral persistence in the natural host, the squirrel monkey.
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CITATION STYLE
APA
Knappe, A., Hiller, C., Niphuis, H., Fossiez, F., Thurau, M., Wittmann, S., … Fickenscher, H. (1998). The Interleukin-17 Gene of Herpesvirus Saimiri. Journal of Virology, 72(7), 5797–5801. https://doi.org/10.1128/jvi.72.7.5797-5801.1998
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