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P53 deletion and NrasG12D cooperate for AML

Blood
DOI: 10.1182/blood-2016-11-750547
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Abstract

In this issue of Blood, Zhang et al show the cooperation between P53 deletion and NRASG12D in the development of acute myeloid leukemia (AML). They also shed light on the megakaryocyte-erythroid progenitor (MEP) being the leukemia-initiating cell (LIC) targeted by P53 deletion and NRASG12D cooperation in mice.1

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Vainchenker, W., & Plo, I. (2017, January 19). P53 deletion and NrasG12D cooperate for AML. Blood. American Society of Hematology. https://doi.org/10.1182/blood-2016-11-750547

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