Abstract
Oxidative stress may contribute to many of the pathophysiologic changes that occur after traumatic brain injury (TBI). There are a number of potential sources and mechanisms for oxygen free radical (OFR) production and lipid peroxidation after TBI. In this study, we investigate the time-dependent changes in xanthine oxidase (XO) activity and lipid peroxidation using a focal TBI animal model. We demonstrate that there is an immediate increase in lipid peroxidation by-products and in XO enzyme activity after TBI. © 2005 Elsevier Ltd. All rights reserved.
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Solaroglu, I., Okutan, O., Kaptanoglu, E., Beskonakli, E., & Kilinc, K. (2005). Increased xanthine oxidase activity after traumatic brain injury in rats. Journal of Clinical Neuroscience, 12(3), 273–275. https://doi.org/10.1016/j.jocn.2004.12.002
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