Abstract
Two dogs developed delayed neurological deterioration after rapid correction of severe hyponatremia. Sequential magnetic resonance imaging showed the development of lesions in the thalamus. One dog was necropsied, and the lesions were characterized by myelinolysis with sparing of axons and neurons. The second dog gradually recovered with no detectable neurological deficits. The syndrome seems analogous to central pontine myelinolysis in human beings. Guidelines for correction of hyponatremia to prevent development of myelinolysis are given. © 1994 American College of Veterinary Internal Medicine
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CITATION STYLE
O’Brien, D. P., Kroll, R. A., Johnson, G. C., Covert, S. J., & Nelson, M. J. (1994). Myelinolysis After Correction of Hyponatremia in Two Dogs. Journal of Veterinary Internal Medicine, 8(1), 40–48. https://doi.org/10.1111/j.1939-1676.1994.tb03194.x
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