Ischemic stroke revealing fahr’s syndrome

Abstract

Fahr’s syndrome is defined as the presence of bilateral intracerebral, symmetrical, nonarteriosclerotic calcifications involving the basal ganglia. Their detection during stroke is exceptional. Phosphocalcium metabolism should be investigated with human parathyroid hormone (HPH) dosage. Diagnosis is based on the evaluation of clinical and laboratory data as well as on radiological imaging. Long-term antiepileptic treatment can cause basal ganglia calcification resulting in Fahr’s syndrome. Prognosis is favorable. The correction of phosphocalcium metabolism usually leads to significant improvement.