Abstract
After damage to the endothelium, platelets prevent excessive bleeding by adhering to the exposed ECM. In this issue of Blood, Kim and colleagues1 describe a novel function of TGFBIp that potentiates platelet adhesion and activation, a protein with a previously established role in corneal dystrophies.
Cite
CITATION STYLE
APA
Rowley, J. W., Weyrich, A. S., & Campbell, R. A. (2009, December 10). TGFBIp: More than meets the eye? Blood. American Society of Hematology. https://doi.org/10.1182/blood-2009-09-243881
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