Abstract
In this issue of Blood, Bateman and colleagues identify the ETV6-RUNX1 fusion as a common prenatal event at the origin of t(12;21)(p13;q22) ALL. 1 Additional genetic lesions in the preleukemic clone are required but follow divergent evolutionary pathways toward overt leukemia.
Cite
CITATION STYLE
APA
Müschen, M. (2010, April 29). Genetic relicts from the origin of ALL. Blood. American Society of Hematology. https://doi.org/10.1182/blood-2010-02-268110
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