Abstract
Interleukin-12 was considered to be the driving force behind pathological conditions associated with several autoimmune disease models, including experimental autoimmune encephalomyelitis (EAE) and collagen-induced arthritis, as well as models of ocular inflammation ([1][1]–[4][2]). However, the
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CITATION STYLE
APA
Khader, S. A., & Thirunavukkarasu, S. (2019). The Tale of IL-12 and IL-23: A Paradigm Shift. The Journal of Immunology, 202(3), 629–630. https://doi.org/10.4049/jimmunol.1801603
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