IMMUNOREACTIONS INVOLVING PLATELETS. V. POST-TRANSFUSION PURPURA DUE TO A COMPLEMENT-FIXING ANTIBODY AGAINST A GENETICALLY CONTROLLED PLATELET ANTIGEN. A PROPOSED MECHANISM FOR THROMBOCYTOPENIA AND ITS RELEVANCE IN “AUTOIMMUNITY”*

  • Shulman N
  • Aster R
  • Leitner A
  • et al.
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Abstract

A thrombocytopenic state developing in the absence of any apparent underlying disorder and accompanied by an abundance of megakaryocytes in the bone marrow represents the syndrome of idiopathic thrombocytopenic purpura (ITP).

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Shulman, N. R., Aster, R. H., Leitner, A., & Hiller, M. C. (1961). IMMUNOREACTIONS INVOLVING PLATELETS. V. POST-TRANSFUSION PURPURA DUE TO A COMPLEMENT-FIXING ANTIBODY AGAINST A GENETICALLY CONTROLLED PLATELET ANTIGEN. A PROPOSED MECHANISM FOR THROMBOCYTOPENIA AND ITS RELEVANCE IN “AUTOIMMUNITY”*. Journal of Clinical Investigation, 40(9), 1597–1620. https://doi.org/10.1172/jci104383

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