Abstract
Postictal transient focal neurological deficits, i.e., Todd’s paralysis, at times are correlated with early veins and/or vascular stain angiographically. Radionuclide gamma camera images show that they also may be accompanied by a relative flow hyperperfusion and a cortical rim static image uptake. Using these observations some portion of Todd’s paralysis may be explained as the result of focal epileptic discharges that lead to local vasomotor and/or metabolic changes. The functional arterial venous shunting that results could contribute to cortical ischemia and the subsequent clinical deficits. © 1975 American Heart Association, Inc.
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CITATION STYLE
Yarnell, P. R. (1975). Todd’s paralysis: A cerebrovascular phenomenon? Stroke, 6(3), 301–303. https://doi.org/10.1161/01.STR.6.3.301
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