Abstract
In this issue of Blood,Nelson and colleagues1 and Warsch and colleagues2 report that signal transducers and activators of transcription 5 (STAT5) is an attractive target to circumvent tyrosine kinase inhibitor (TKI) resistance in chronic myeloid leukemia (CML).
Cite
CITATION STYLE
APA
Turhan, A. G. (2011, March 24). STAT5 as a CML target: STATinib therapies? Blood. American Society of Hematology. https://doi.org/10.1182/blood-2011-01-332569
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